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Difference between revisions of "Talk:O2k-Publications: Topics"

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* 2017-06-08 '''Patent of Q-electrode (1988)''': Dr P.R. Rich, Glynn Res. Ph., Bodmin; European Patent no.85900699.1 [[User:Hungerm|Hungerm]] ([[User talk:Hungerm|talk]]) 10:18, 21 June 2017 (CEST)  
* 2017-06-08 '''Patent of Q-electrode (1988)''': Dr P.R. Rich, Glynn Res. Ph., Bodmin; European Patent no.85900699.1 [[User:Hungerm|Hungerm]] ([[User talk:Hungerm|talk]]) 10:18, 21 June 2017 (CEST)  


* 2017-06-08 ''' Pre-print publication: Mitochondria are physiologically maintained at close to 50 °C [http://www.bioblast.at/index.php/Chretien_2017_bioRxiv »Bioblast link«]
* 2017-06-08 ''' Pre-print publication: Mitochondria are physiologically maintained at close to 50 °C [[Chretien_2017_bioRxiv |»Bioblast link«]]


* 2017-05-30 '''Quote of the day''': ".. oligomycin (complex V inhibitor) determines the amount of ATP production is linked to respiration, FCCP (mitochondrial uncoupler) enables measurement of maximal respiratory capacity, while a combination of rotenone (complex I inhibitor) and antimycin A (complex III inhibitor) determines the reserved respiratory capacity." .. and more: "The mitochondrial network is a major site of ATP production through the coupled integration of the electron transport chain (ETC) with oxidative phosphorylation." - Trotta et al (2017) Disruption of mitochondrial electron transport chain function potentiates the pro-apoptotic effects of MAPK inhibition. JBC Papers in Press. Published on May 25, 2017 as Manuscript M117.786442.
* 2017-05-30 '''Quote of the day''': ".. oligomycin (complex V inhibitor) determines the amount of ATP production is linked to respiration, FCCP (mitochondrial uncoupler) enables measurement of maximal respiratory capacity, while a combination of rotenone (complex I inhibitor) and antimycin A (complex III inhibitor) determines the reserved respiratory capacity." .. and more: "The mitochondrial network is a major site of ATP production through the coupled integration of the electron transport chain (ETC) with oxidative phosphorylation." - Trotta et al (2017) Disruption of mitochondrial electron transport chain function potentiates the pro-apoptotic effects of MAPK inhibition. JBC Papers in Press. Published on May 25, 2017 as Manuscript M117.786442.

Revision as of 08:32, 22 June 2017

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  • 2017-06-08 Patent of Q-electrode (1988): Dr P.R. Rich, Glynn Res. Ph., Bodmin; European Patent no.85900699.1 Hungerm (talk) 10:18, 21 June 2017 (CEST)
  • 2017-06-08 Pre-print publication: Mitochondria are physiologically maintained at close to 50 °C »Bioblast link«
  • 2017-05-30 Quote of the day: ".. oligomycin (complex V inhibitor) determines the amount of ATP production is linked to respiration, FCCP (mitochondrial uncoupler) enables measurement of maximal respiratory capacity, while a combination of rotenone (complex I inhibitor) and antimycin A (complex III inhibitor) determines the reserved respiratory capacity." .. and more: "The mitochondrial network is a major site of ATP production through the coupled integration of the electron transport chain (ETC) with oxidative phosphorylation." - Trotta et al (2017) Disruption of mitochondrial electron transport chain function potentiates the pro-apoptotic effects of MAPK inhibition. JBC Papers in Press. Published on May 25, 2017 as Manuscript M117.786442.
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