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Difference between revisions of "Rasmussen 2003 Eur J Physiol"

From Bioblast
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|enzymes=Complex I, Complex II;succinate dehydrogenase, Complex V;ATP synthase
|enzymes=Complex I, Complex II;succinate dehydrogenase, Complex V;ATP synthase
|diseases=Aging;senescence
|diseases=Aging;senescence
|topics=Substrate; Glucose; TCA Cycle
|topics=Substrate;glucose;TCA cycle
}}
}}
__TOC__
__TOC__

Revision as of 10:45, 25 February 2015

Publications in the MiPMap
Rasmussen UF, Krustrup P, Kjaer M, Rasmussen HN (2003) Human skeletal muscle mitochondrial metabolism in youth and senescence: no signs of functional changes in ATP formation and mitochondrial oxidative capacity. Pflugers Arch – Eur J Physiol 446:270-78.


Rasmussen UF, Krustrup P, Kjaer M, Rasmussen HN (2003) Eur J Physiol

Abstract:


Labels: Pathology: Aging;senescence 

Organism: Human  Tissue;cell: Skeletal muscle  Preparation: Isolated mitochondria  Enzyme: Complex I, Complex II;succinate dehydrogenase, Complex V;ATP synthase  Regulation: Substrate;glucose;TCA cycle"Substrate;glucose;TCA cycle" is not in the list (Aerobic glycolysis, ADP, ATP, ATP production, AMP, Calcium, Coupling efficiency;uncoupling, Cyt c, Flux control, Inhibitor, ...) of allowed values for the "Respiration and regulation" property. 




References

Both muscle strength and peak contraction velocity decline, in some muscles even from the age of 20

[8, 15, 16 17, 22, 23, 26, 27, 28, 29, 30, 32, 36, 37]

The response to training is independent of age

[8, 9, 15, 17, 22, 23, 30, 32, 36, 37].

Loss of type-II fibres is greater than that of type-I

[15, 16, 22, 23, 26, 27, 28, 37].

Cytochrome oxidase deficient fibres appear later in increasing, but very small numbers

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